Colorectal cancer is killing more 20- to 30-year-olds. We have some clues about why. – VOX

Colorectal cancer is killing more 20- to 30-year-olds. We have some clues about why. – VOX

The latest national cancer statistics from the American Cancer Society show adults under the age of 55 have seen their colorectal cancer rate increase 2 percent per year since the mid-1990s. Overall, those born in 1990 have double the risk of developing colon cancer and four times the risk of getting rectal cancer compared to those born around 1950. That’s why, in response to the alarming trend, the ACS in 2018 lowered the recommended age for routine colorectal cancer screening to 45 from 50.

“It’s not like the problem is bad and has stabilized,” said Thomas Weber, the director of surgical oncology at New York’s Northwell Health, who organizes an annual summit for researchers trying to solve the mystery. “The problem has continued to worsen.”

via Causes of colorectal cancer: obesity may play a role – Vox

They might want to look in the direction of heavy metal radioactive poisons, GMO foods, chemicals and vaccine negative side effects.

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    Dr. Busbys article

    Open Access

    A Risk Coefficient for Radiation-Induced Dementia

    Open Access Maybe reprinted
    DOI: 10.4236/aad.2018.72002 475 Downloads 1,496 Views
    Author(s) Leave a comment Christopher Busby Affiliation(s)
    Environmental Research SIA, 1117 Latvian Academy of Sciences, Riga, Latvija.
    The effect of ionising radiation exposure on dementia is approached by applying the causation models of John Stuart Mill and of Sir Austin Bradford Hill to mechanism and epidemiological evidence. Since ionizing radiation is known to kill brain cells in laboratory culture and to affect hippocampal neurogenesis in animal experiments at modest doses, it is reasonable to assume that exposure to radiation must affect neurological integrity and hence dementia rates in those who are exposed. There is persuasive evidence from the epidemiological studies of a large cohort of female nuclear workers that ionising radiation exposure is associated with significant low dose region dose-dependent increases in rates of dementia. Using results from these studies, the Probability of Causation approach (PC), conventionally employed for assessing cancer risk following radiation exposure, is extended to dementia to find a risk coefficient for all ages of 60 per Sievert cumulative exposure over the range 0-100 mSv. The finding suggests that natural background external exposures to ionizing radiation are partly responsible for the development of dementia in human populations. A simple general model for dementia is proposed.
    Radiation, Alzheimer, Dementia

    In response to Mining Awareness, which in my estimation is one of the best blogs on the internet with and A Green Road

    “Miningawareness on April 19, 2019
    In response to the above comment w, I continue to wonder if Kuru, and the related Mad Cow, CJD, and CWE for deer aren’t all caused by radioactive materials/fallout. That would account for the fact that a disease causing agent can’t be found and that it supposedly sneaks around the immune system. It explains many other oddities, too.
    Traditional CJD is like a fast moving Alzheimers.”

    This was in response to an article posted on a study by Dr Busby linking radionuclides to dementia

    Six Points

    1. Problems of absorption of CJD Kuru and CWE proteina
    Kuru, CJD, and CWE are supposed to be linear proteins which are amino acids held togethe by peeptide bonds.
    Peptide bonds are very suscwptible to  breaking by stomach acid and digestive enzyme.  That is why insulin has to be injected.

    2. Large proteins like these prions, do not pass the blood-brain barrier easily. They are helical structures. They also contain polar disulfide bonds which do not allow them to pass lipophillic tissue or membranes easily.

    3. The mechanism of action of the prion proteins, is supposed to based on thier abilitity to make cells generate amyloid alzeimers-like plaques in the brain. Abnormal proteins are supposedly manufactured in cells, effected by prions. The abnormal proteins build up in between cells, to form amyloid plaque deposits, that cause dementia.

    There is not a clear mechansim of action whereby the prions regenerate themselve or, force cell nuclei to generate the aforementioned plaques.

    4. Strong radionuclides like Cesium 137 act like electrolytes found in brain tissue and are easily accessible to the brain.
    Cesium 137 acts like potassium. It is easily accessible to brain tissue.

    Cesium 137 would damage and dna and disrupt protein sythesis in cells. it would do so, by damaging RNA. This would disrupt protein sythesis and cause abnormal amyloid protein synthesis. Much more plausible than the prion theory.
    Strong salt like radionuclides, would also effect the hydrogen bonding, essential for proper protein synthesis and transport in cells.

    The same could be said for Strontium 90, which would also have high affinity for nerve tissue
    like cesium 137. These highly potent gamma and beta emitter would also cause cell-death.

    Plutonium is mistaken for iron, by the body.  It is found in neural tissue.
    Plutonium and transuranics are strong alpha emitters.

    Plutonium’s actions actions would be more insidious . 
    Plutonium and other heavy metal-like, radionuclides could do mulitiple damage to nerve cell tissue and glial cell tissue in the brain.

    The damage from Plutonium, includes the generation of amyloid plaques, that would contribute to alzeimers dementias and other dementias from cell death.
    it would  get stuck in glial cells, lipose tissue, and nerve tissue.

    Plutonium would  constantly degrade dna and rna process in the brain and kills brain cells as Dr. Busby, alludes to in his paper on radionuclides and demential. It would sevelerly damage hippocampal tissue, where new brains cells are made, as Dr Busby says.

    Radionuclides like tritium and carbon-14 would have easy acces to brain tissue, to create destructive environments in nerve and glial cells. They would also bombard rna and effect protein synthesis.

    5. There was a great deal of Nuclear Bomb testing in the south pacific before the Kuru was suppisedly discovered. There could also be a comorbidity where the radionuclides enhanced the toxicity or actually are responsible for the appearance of prion like plaques in the brain.

    6. The CWE  and CJD have appeared well into the nuclear age with the glut of radionuclide pollution and fallout that began with uranium mining , open air testing, nuclear reactor pollution, nuclear reactor meltdowns and nuclear waste accumulation.


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